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Home Supplements/Vitamins Immune Function Coenzyme Q10 30 Tablets - NRF
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Coenzyme Q10 30 Tablets - NRF
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Coenzyme Q10 30 Tablets - NRF

Price: R249.00

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Coenzyme Q10 tablets provide a high potency scientific formulation of the antioxidant Coenzyme Q10, in a bioavailability enhancing FoodMatrixTM base.


  • Cardiovascular disease.
  • Cancer.
  • Weight loss aid.
  • Muscular dystrophy.

Composition per daily dose:

Item  Quantity % RDA
Coenzyme Q10 30 mg  *
Food Extract       
FoodMatrixTM 300 mg  *

         FoodMatrixTM formulated in a food carrier base of plant carotenoids, protein (amino acids) and carbohydrates

      * % RDA not established

Dosage and Directions for use:

  • Take one (1) tablet daily, before meals or as directed by your healthcare practitioner.

Potential Benefits:

  • Increased energy.
  • Facilitates weight-loss.
  • Improves antioxidant status.
  • Reduces the risk of cancer.

Mechanism of action:

    The mitochondria has appropriately been termed the "powerhouse" of the cell, since it is within this organelle that most of the capture of energy, derived from respiratory oxidation, takes place. The system in mitochondria that couples respiration to the generation of high-energy intermediate, ATP, is termed oxidative phosphorylation.

    Oxidative phosphorylation enables aerobic organisms to capture a far greater portion of the available free energy of respiratory substrates compared with anaerobic organisms. The chemiosmotic theory offers an insight into how this is accomplished. A number of drugs and poisons (e.g. Carbon monoxide) inhibit oxidative phosphorylation, usually with near fatal consequences. Additionally a number of inherited defects of mitochondria involving components of the respiratory chain and oxidative phosphorylation have been reported. Patients present with myopathy, encephalopathy, and often have lactacidosis.

    All of the useful energy liberated during oxidative phosphorylation of fatty acids and amino acids and nearly all of that released from the oxidation of carbohydrates are made available within the mitochondria as reducing equivalents (free radicals). The mitochondria contain the series of catalysts known as the respiratory chain that collect and transport reducing equivalents and direct them to their final reaction with oxygen to form water. Also present is the machinery for trapping the liberated free energy as high energy-phosphate. The mitochondria also contain the enzyme systems responsible for producing most of the reducing equivalents in the first place, i.e., the enzymes of oxidation and of the citric acid cycle. The latter is the final common metabolic pathway for the oxidation of all the major foodstuffs.

    In recent years, it has become clear that an additional carrier is present in the respiratory chain linking the flavoproteins to cytochrome b, the member of the cytochrome chain of the lowest redox potential. This substance known as ubiquinone (Coenzyme Q10) exists in the mitochondria in the oxidised quinone form under aerobic conditions and in the reduced quinol form under anaerobic conditions. Coenzyme Q10 is a constituent of the mitochondrial lipids; the other lipids are predominantly phospholipids that constitute part of the mitochondrial membrane. Under normal circumstances Coenzyme Q10 is present in a large stoichiometric excess, compared to the other members of the respiratory chain; this suggests that Coenzyme Q10 is a mobile component of the respiratory chain and that it collects reducing equivalents (free radicals), neutralises and passes them on to the cytochromes.

Numerous studies have shown the efficacy of Coenzyme Q10 supplementation in the following conditions:

  • Aids/HIV: The particular article used as reference reported on two patients, who have survived four to five years, with no symptoms of adenopathy or infection while continuously taking Coenzyme Q10. Additionally the authors reported results on 14 normal subjects that experienced increased T4/T8 ratios in response to Coenzyme Q10 administration.2
  • Cancer: The quoted article sites numerous studies and clinical trials supporting the case of Coenzyme Q10 supplementation in the treatment of breast cancer.3
  • Cardiovascular / Coronary Heart Disease: The effects of Coenzyme Q10 administration on stable angina pectoris patients showed the efficacy and safety of Coenzyme Q10 supplementation.4 A further study showed that Coenzyme Q10 supplementation reduced the mortality rate in patients suffering from cardiomyopathy, and significantly increased their survival rates.5
  • Diabetes: A 70 year old man diagnosed with diabetic amyothrophy, experienced relief in symptoms of the legs, fatigue and residual urine in bladder following Coenzyme Q10 supplementation.6
  • Immune enhancement: Coenzyme Q10 has been cited by numerous studies to be a key factor in optimal immune functioning.7
  • Liver damage: Early studies indicate the effectiveness of Coenzyme Q10 in preventing hepatotoxicity in rats.8
  • Lung disease: results from the quoted study showed that the oral administration of Coenzyme Q10 had significant beneficial effects on muscular energy metabolism in chronic lung disease patients suffering from hypoxemia either during exercise or rest.9


   1. Murray RK, Granner DK, Mayes PA, Rodwell VW. Harper’s Biochemistry – 22nd Edition. Prentice – Hall Internationals Inc. p. 112.
   2. Folkers K, et all. Coenzyme Q10 increases T4/T8 ratios of lymphocytes in ordinary subjects and relevance to patients having HIV related complex. Biochem. Biophys Res. Commun, April 30, 1991; 176(2): 786 – 791.
   3. Lockwood K, et all. Progress on therapy of breast cancer with vitamin Q10 and the regression of metastases. Biochem. Biophys Res. Commun, July 6, 1995; 212(1): 172 – 177.
   4. Kamikawa T, et all. The effects of Coenzyme Q10 on exercise tolerance in chronic stable angina pectoris. American Journal of Cardiology, August 1, 1985; 56(4): 247 – 251.
   5. Langsjoen PH, et all. Pronounced increase of survival of patients with cardiomyopathy when treated with Coenzyme Q10 and conventional therapy. Int. J. of Tissue React. 1990; 12(3): 163 – 168.
   6. Suzuki Y, et all. A case of diabetic amyotrophy associated with mitochondrial tRNA mutation and successful therapy with Coenzyme Q10. Endocr. Journal, April 1995; 42(2): 141 – 145.
   7. Folkers K, Wolaniuk A. Research on Coenzyme Q10 in clinical medicine and immunomodulation. Drugs Exp. Clin. Research. 1985; 11(8): 539 – 545.
   8. Yoshikawa T, et all. The protection of Coenzyme Q10 against carbon tetrachloride hepatotoxicity. Gastroenterol. Jpn. 1981; 16(3): 281 – 285.
   9. Fujimoto S, et all. Effects of Coenzyme Q10 administration on pulmonary function and exercise performance in patients with chronic lung disease. Clinical Investigations (8 Suppl) 1993; 71: S162 – S166.
  10. Hole John, W Jnr. Human Anatomy Physiology 6TH Edition. Wm. C. Brown Publishers, Dubuque, Iowa, Melbourne – Australia, Oxford – London.
  11. Cicoke AJ. Enzymes and Enzyme Therapy. New Canaan, CT; Keats Publishing, 1994.

Special precautions and contra-indications:

  • None known.

Potential Drug Interactions:

  • None known.

Contains no preservatives, artificial colouring and flavouring.

Gluten free.


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Last Updated: Thursday, 19 September 2019 06:29

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